Cortical plasticity in episodic and chronic cluste in Research & Scientific News Posted November 26, 2014 And there are two of the biggest barriers we have faced in the history of CH. First, we look to the "renowned researchers" and put them on a pedestal, waiting for them to hand down their wisdom and treating only their words as valid. We can all go ahead and name them, Goadsby and Sewell and so on and so forth. . .I'll even throw in hacks like Halpern. We canonize them based on what, exactly? I can appreciate their work, but they are not gods, nor should they be the only ones whose work is considered valid. Second, the old hypothalamus thing again. It's a little like quantum physics, really. If you fixate on one thing for long enough, your observations will confirm everything you see, and that will be your "fact". So for years, we have been stating, without doubt, that the hypothalamus is the root cause of CH. But what does that particle do when you're not staring at it? What if we've been staring at the hypothalamus for so long that it has caused us to form incorrect hypotheses about CH? I don't mean to bust anybody's balls here, but I see a lot of cognitive dissonance when it comes to CH. We hold these things as facts because we can't make ourselves believe otherwise. It's hard to point the finger at something we can't see, medicate, or surgically remove. But let's face it, the mechanism behind CH meets all three of those criteria, doesn't it?